Diagnosis and prevention of chicken gout

Chicken gout is uricemia caused by disturbance of protein metabolism.

First, etiological analysis.

1. Nutritional imbalance: when the content of protein and nuclear protein in the feed is too high, the sharp increase of uric acid in the body exceeds the excretion limit, thus depositing in the form of urate (mainly sodium urate) in the joint, visceral surface and subcutaneous connective tissue, causing a series of pathological changes. Especially in chickens with imbalance of calcium and phosphorus in feed and vitamin An and D deficiency, the disease is more likely to occur.

two。 Chicken renal dysfunction: when feeding too many sulfonamides and other factors caused by renal dysfunction, so that the excretion of uric acid is blocked and accumulated, can cause gout.

Second, the incidence and symptoms.

The disease is non-seasonal, mostly group-style, and is generally chronic. The diseased chicken showed systemic nutritional disorder, loss of appetite, gradual weight loss, mental malaise, loose feathers, anemia, pale crown, periodic increase in body temperature, rapid heartbeat, white semi-mucous thin feces discharged from the anus, and a large amount of urate in the feces. The egg production of hens decreased or even stopped completely. Individual chickens have difficulty breathing and even have neurological symptoms such as spasms, and most of them die of exhaustion. Joint gout is characterized by swelling of the joints of the toes and legs, slow movement, lameness, and difficulty standing.

Third, pathological changes were examined by autopsy.

During the autopsy, the kidney was obviously enlarged, the color became lighter, and there were white spots formed by urate deposition on the surface. In severely diseased chickens, this calcareous urate deposit is often covered on the surface of other internal organs (liver, heart, spleen) and mesentery, and many of them can form a white film. Joint gout autopsy showed white urate deposition on the joint surface and tissue around the joint, and erosion occurred on some joint surfaces. When the sediment is scraped down and observed under a microscope, many acicular crystals of sodium urate can be seen.

Fourth, diagnosis.

The incidence and clinical symptoms of chicken gout are easy to be confused with other diseases. Chicken gout should be diagnosed according to the characteristic pathological changes of pathological examination, feeding investigation and etiological analysis.

Fifth, prevention and control.

1. Promote the use of full-price feed to prevent malnutrition. Farmers' own feed must be designed according to the requirements of different breeds, different development stages and different seasons, and the nutritious feed must be prepared. To prevent the blind addition of fish meal and other animal protein feed, especially in winter should pay attention to the right amount of vitamin A, D.

two。 When preventing and controlling chicken diseases, use sulfonamides, sodium bicarbonate and other drugs, not for too long, but also to prevent excessive, or stop using when gout is suspected.

3. Overcrowded chicken houses, lack of exercise and lack of light, damp and cold poultry houses, as well as coccidiosis, chicken pullorum, Newcastle disease and other can affect the normal function of the kidney, can promote the occurrence of the disease. Therefore, it is necessary to control the feeding environment, strengthen feeding management, and do a good job in the comprehensive prevention and control of the epidemic disease in peacetime, which can reduce the occurrence of the disease.

4. In order to reduce kidney damage, sulfonamides should be used or not used as little as possible in the process of prevention and treatment of chicken diseases, and adequate drinking water should be maintained around the clock to ensure the excretion of harmful toxins in the kidneys.