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How to prevent and cure leptospirosis (porcine dysentery)?

Published: 2024-11-05 Author: mysheen
Last Updated: 2024/11/05, Porcine dysentery is an acute infectious disease caused by Treponema pallidum, which is characterized by different degrees of mucinous hemorrhagic dysentery. The disease can occur in all seasons of the year, and can occur in pigs of all ages, but the incidence is high in piglets aged 2-3 months. The most acute type of the disease can suddenly occur and die, which is also a sign that the disease will break out in pigs. Acute symptoms followed. At the beginning of the disease, the spirit is slightly poor, the appetite is reduced, the feces become hard, and there are strips of mucus on the surface. After that, he quickly developed dysentery. Faeces are yellow, soft or watery. Severe cases were faeced for 2 days in 1 Mel.

Porcine dysentery is an acute infectious disease caused by Treponema pallidum, which is characterized by different degrees of mucinous hemorrhagic dysentery. The disease can occur in all seasons of the year, and can occur in pigs of all ages, but the incidence is high in piglets aged 2-3 months. The most acute type of the disease can suddenly occur and die, which is also a sign that the disease will break out in pigs. Acute symptoms followed. At the beginning of the disease, the spirit is slightly poor, the appetite is reduced, the feces become hard, and there are strips of mucus on the surface. After that, he quickly developed dysentery. Faeces are yellow, soft or watery. In severe cases, the stool was filled with blood and mucus, or mixed with blood clots, for 2 days. At the same time of dysentery, the body temperature was slightly higher (about 40.5 ℃), maintained for several days, and then decreased to normal. The body temperature dropped below normal before death. With the development of the course of the disease, the diseased pigs were depressed, lost weight, increased thirst, stench of feces, blood, mucus and fragments of necrotic epithelial tissue. The sick pig gradually lost weight, the skin was pale, the abdomen was sunken, stood up powerless, was extremely weak, and finally died.

The subacute type had mild condition, more dysentery, more mucous membrane and necrotic tissue fragments, less blood and longer disease period. Progressive emaciation and growth stagnation. Although the mortality rate is not high, it has poor growth and development, and the feed reward is reduced, which has a great impact on production.

Autopsy showed congestion and edema of the large intestine, especially of the colon wall and mesentery, and mesenteric lymphoid enlargement. The surface of the intestinal mucosa was often covered with gelatinous mucus and fibrinous exudates with blood clots.

Prevention and treatment:

Clenbuterol, metronidazole, lincomycin, intramuscular injection or oral administration, once a day, for 5 days as a course of treatment. Drugs should be used alternately to prevent the development of drug resistance.

 
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