Chicken infectious anemia-symptoms
Under natural conditions, the incubation period of this disease is not very clear, but the earliest 12 days can show clinical symptoms, the third to fourth weeks death increased. Anaemia and pathological changes of tissues appeared 8 days after artificial infection of 1-day-old chicks, and the disease began from 10 to 14 days, and then died.
Anemia is a characteristic symptom of the disease. The anemia of infected chickens was most serious 14 - 16 days after infection, and the hematocrit value dropped below 20%. Infected chickens show anorexia, depression, weakness, emaciation, weight loss, pale beaks, whiskers and visible mucous membranes, subcutaneous and muscle bleeding, and bleeding from wing tips is also very common. The number of red blood cells and white blood cells decreased to 109/L and 5×106/L respectively. A large number of chickens died 5 days to 6 days after infection, and the mortality rate was usually less than 30%. The chickens survived 20 days to 28 days after infection gradually recovered to health, but most of them grew slowly and became stiff chickens. If secondary bacterial, fungal or viral infection, can aggravate the disease, hinder recovery, death increased.
CIAV can cause immunosuppression, so infected chickens often secondary Clostridium perfringens and Staphylococcus aureus infection, causing gangrenous dermatitis of muscle and subcutaneous tissue; also can make chickens to Escherichia coli, inclusion body hepatitis adenovirus, infectious bursal disease virus, Marek's disease virus and reovirus and other pathogens susceptibility increased. In some flocks, a second death peak occurred two weeks after the first death peak, which was often caused by secondary infection in addition to horizontal transmission.
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Treatment of calcium and phosphorus Metabolism Disorder in Pigs with combination of traditional Chinese and Western Medicine
Calcium and phosphorus metabolic disorders occur in young pigs, resulting in bone dystrophy called rickets. The pathological features were insufficient calcification of osteoblasts and temporary calcification of persistent cartilage hypertrophy and epiphyseal enlargement. The clinical features are digestive disorder, heterophobia, lameness and bone deformation. It is a non-inflammatory disease of bone tissue dysplasia. Vitamin D deficiency also plays an important role in the pathogenesis of rickets. Calcium and phosphorus metabolic disorders occur in adult pigs, resulting in osteodystrophy called osteomalacia, or fibrous osteodystrophy. The pathological feature is the progression of bone
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Causes of nutritional and metabolic diseases in chickens
1. Insufficient intake of nutrients; insufficient supply of ① diets; lack of protein (especially essential amino acids), fat, vitamins, mineral trace elements and other nutrients in ② diets; insufficient intake of nutrients caused by ③ lack of appetite. 2. the demand for nutrients increases, but it is not supplied in time, for example, under pathological conditions, the consumption of nutrients in the body increases. Nutritional and metabolic diseases such as febrile diseases, parasitic diseases and diarrhoea. 3. Nutrient digestion, malabsorption of gastrointestinal tract, liver, kidney, pancreas and other dysfunction
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