Prevention of Fatty liver in Perinatal Dairy cows without Rumen Movement

In winter, the perinatal cows with fatty liver raised in warm shed may suffer from insufficient bile secretion due to liver function disorder, affecting digestive function; at the same time, it is easy to secondary other diseases such as retained placenta, production paralysis, endometritis, etc., thus affecting the milk yield, fertility and immunity of sick cows. The disease incidence of 5~9 years old dairy cows was the highest, and that of the first-born dairy cows was low.

cows

Cause: Improper feeding management. After the cow gives birth, the sugar and other nutrients in the body are continuously excreted with the milk. If not timely supplement nutrition, resulting in negative energy balance, cows to provide energy for the tissue will have to use the body fat reserves. Because of excessive free fatty acids entering the liver or hypoglycemia in cattle, the ability of liver tissue to clear very low density lipoprotein decreases, and the protein transport out of the liver is blocked and fatty liver is formed. Feeding too much concentrate can also cause the disease.

Endocrine dysfunction: dairy cows are affected by pregnancy, childbirth, lactation and other factors, so that the pituitary gland, adrenal gland overload. Because adrenal gland function is not complete, cause sugar xenogenesis to reduce, and rumen to glycogen utilization occurrence obstacle, make blood sugar drops and come on ill. After delivery, protein-bound iodine significantly reduced, resulting in thyroid insufficiency, but also fatty liver.

Other secondary disease effects. Some consumptive diseases of dairy cows, such as atony of the forestomach, traumatic gastritis, abomasum displacement, osteomalacia, production paralysis and other chronic infectious diseases, can be secondary to fatty liver.

Symptoms: affected cattle refused to feed concentrate, silage, and may appear pica, thin body, subcutaneous fat disappeared, skin elasticity weakened. Dry and hard feces, severe, with loose stools. Sick cattle are moderately depressed, unwilling to walk, feed, and sometimes have mild abdominal pain. General temperature, pulse, respiration normal, rumen movement weakened, long course, rumen movement disappeared. Cows with severe fatty liver can die of excessive weakness or poisoning and other accompanying diseases if they do not receive timely and correct treatment; those suffering from mild and moderate fatty liver can heal themselves in about one and a half months, but milk production cannot be completely restored, and immunity and fertility are affected.

Control methods:

Strengthen feeding management and supply nutrition reasonably. For pregnant cows, concentrate feeding should be appropriately reduced to avoid prenatal over-fertilization; pregnant diet should contain sufficient cobalt, phosphorus and iodine, and increase exercise amount appropriately in late pregnancy; postpartum cows should strengthen nursing, improve diet palatability, gradually increase concentrate, avoid energy negative balance caused by postpartum lactation. At the same time, it is necessary to treat gastrointestinal diseases affecting digestion and absorption in time.

Glucose injection therapy. 500 ml 50% glucose solution was injected intravenously once a day for 4 days. 1000 ml of 20% glucose solution can also be injected intraperitoneally. At the same time, betazone 20 mg im, and oral intake with feed 250 ml propylene glycol or glycerol, twice a day, for 2 days. Then change to 110 ml per day, and then take 3 days, the effect is better.

Oral niacin, choline. From the 14th day before birth, each cow was fed 8 g nicotinic acid, 80 g choline chloride and 60 g cellulase every day. If it could be combined with intravenous injection of high concentration glucose solution, the effect would be better.