Broiler ascites syndrome

Broiler ascites syndrome is also known as "heart failure syndrome"; also known as broiler pulmonary hypertension syndrome; abdominal enlargement, fluctuating touch, thinning and shining abdominal skin, severe redness. Autopsy examination showed that there were a large number of yellowish or clear and transparent fluid in the abdominal cavity, some mixed with cellulose deposits, heart failure, pericardial effusion and liver and kidney lesions, mainly occurred in broilers. The disease not only has a high fatality rate, but also affects the feeding efficiency because of reducing the slaughtering grade of broilers, which does great harm to the production of broilers. Because of its complex etiology and pathogenesis, once the disease occurs, it is difficult to control. At present, there is no special treatment. Only aiming at the etiology and taking comprehensive prevention and control measures can the occurrence and loss of ascites be reduced. First, the cause of the disease: the fast-growing broilers with inherent sexual factors have higher requirements for energy and oxygen, and the red blood cells of broilers are larger than those of laying hens, especially during the period of rapid growth within 4 weeks of age. Red blood cells can not flow smoothly in the pulmonary capillaries, affecting blood perfusion in the lungs, leading to pulmonary hypertension and subsequent right ventricular failure. 2. Primary factor 1) chronic hypoxia: broilers raised at high elevations have low oxygen pressure due to thin air. The broiler houses raised in low altitude and sea level areas are not well ventilated, especially when the doors and windows are closed in winter, the concentration of carbon dioxide, ammonia, carbon monoxide and dust in the chicken house increases, and the oxygen pressure decreases, resulting in chronic hypoxia in the body. this leads to pulmonary capillary thickening, stenosis, elevated pulmonary artery pressure, followed by right ventricular enlargement and failure. 2) High-energy diet or pellet feed: high-energy diet or pellet feed can increase the feed intake of broilers and increase the energy and oxygen demand of broilers. 3. Secondary factors 1) nutritional stress: deficiency or excess of some nutrients caused secondary ascites on the basis of primary disease, such as selenium, VE, VC deficiency, salt excess, etc., but it was clear in the mid-1980s that ascites was not caused by a single nutrient deficiency. Some experimental chickens did not find evidence of selenium or VE deficiency, and the addition of VC and minerals in feed could not reduce the incidence of ascites. 4. Others: respiratory diseases or air-source pollutants lead to dysfunction of lung gas exchange, which in turn leads to an increase in pulmonary vascular blood pressure. It is generally believed that lung disease is the initial focus of ascites. In the flock of broilers with ascites, almost no chicken has normal and healthy lungs. 2. Pathological changes 1. The clinical symptoms are sudden death, usually the sick chicken is smaller than the normal chicken, and the feathers are dishevelled and tired, the sick chicken is not willing to move, dyspnea and cyanosis. The most obvious clinical symptoms visible to the naked eye are the enlarged abdomen of the diseased chicken, showing a water bag, fluctuating touch pressure, and thin and shiny abdominal skin. In severe cases, the skin is red and bruised, and some sick chickens have difficulty standing, landing on their abdomen in the shape of a penguin, slow and duck-like. The abdominal cavity was punctured to discharge a transparent and clear yellowish liquid. 3. Preventive measures: 1. To improve the feeding and management of broilers, the early growth rate is fast, the sensitivity to ascites is high, and the effect of early feed restriction is obvious. First, the incidence of ascites in broilers fed with low-nutrition diets was much lower than that of broilers fed with high-nutrition diets. It is suggested that low-energy diets should be fed before 3 weeks of age, and then converted to high-energy diets. Second, feeding powder can effectively control ascites in broilers. Try not to add fat to the diet of broilers in areas prone to ascites. Are selenium, VE and VC panacea? It is said that adding these things can reduce the incidence of disease. Looking forward to the answer to ing. When diseases occur in chickens, it is very important to add vitamins as an auxiliary measure for treatment, especially VA, Vc and VK. Some studies have shown that VE and Vc can enhance the immune function of the body, improve the tolerance of chickens to various stresses, and promote post-disease recovery and growth and development. VK can shorten clotting time and reduce blood loss, so it can relieve symptoms and reduce death in some diseases with bleeding symptoms. It seems that adding some acidifiers to the feed can reduce the occurrence of ascites. Give it a try? Reply to cheng_zhang 's good thank you suggestions. I hope the friends above will also see your plan!