MySheen

Spring is the season of onset, which can be secondary emphysema

Published: 2024-09-19 Author: mysheen
Last Updated: 2024/09/19, Beijing and many areas in the north, last winter without snow, until recently, warm spring flowers suddenly change the climate, cold wind, rain and snow, lasting for many days. Weak constitution, sub-health, old and young groups, etc. are prone to colds, calls...

In Beijing and many areas in the north, there was no snow last winter, until recently, the climate suddenly changed suddenly in the warm spring, with cold winds, rain and snow, stretching for many days. Physical weakness, sub-health, old and young groups are very prone to colds, upper respiratory tract inflammation, lung inflammation, these inflammation are easy to be regarded as colds and ignore treatment, some patients who have already suffered from respiratory diseases, it is also easy to relapse in rapid climate changes. If you ignore treatment, it will become chronic in the development, or complicated lung disease, emphysema is one of the complications.

Cold

Emphysema is mostly secondary and concomitant diseases, a variety of chronic diseases of the lung, such as bronchial asthma, chronic bronchitis, bronchiectasis, etc., not timely or thorough treatment, are easy to develop into emphysema. So in the category, emphysema also has paraseptal emphysema, compensatory emphysema, senile emphysema, interstitial emphysema, obstructive emphysema, focal emphysema and so on.

Obstructive emphysema

After a variety of chronic pulmonary diseases complicated with emphysema, it is usually in the acute stage, and it will enter the stable stage and recovery stage after treatment. if the treatment is not proper, it is easy to relapse from the stable and recovery stage to the aggravating stage. In chest X-ray, CT and other findings, we can see the destruction of the air wall in the distal end of pulmonary terminal bronchioles, such as alveoli, bronchioles, alveolar tubes, alveolar sacs and so on. There will also be different specific manifestations due to differences in obstruction, compensation, interstitial and so on.

Bronchitis

Clinically, the most common is the neglect of treatment after aggravation of senile bronchitis, or long-term direct or indirect smoking, stimulating bronchial wall mucosa and hyperemia, edema, and secreting more mucus, and then the wall is getting thicker and thicker in self-repair. Mucous membrane epithelial cilia because of adhesion, lodging, it is difficult to discharge mucus, so that mucus accumulates more and more, the lumen becomes narrower or blocked, bronchioles dilate when inhaling, air enters alveoli, bronchioles shrink and collapse when exhaling, gas is difficult to exhale, alveoli become emphysema due to excessive inflation or even expansion or rupture.

 
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