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Treatment of vitamin A deficiency in farmed foxes

Published: 2024-11-05 Author: mysheen
Last Updated: 2024/11/05, [etiology] Vitamin An in feed does not meet the requirements of foxes, or vitamin An in diet is destroyed due to excessive storage, oxidation, corruption and improper preparation, or the absorption of vitamin An is affected by digestive tract diseases, it is the main cause of this disease.

[etiology] Vitamin An in feed can not meet the requirements of foxes, or vitamin An in diet is destroyed due to long storage, oxidation, corruption and improper preparation, or the absorption of vitamin An is affected by digestive tract diseases. is the main cause of the disease.

[symptoms] the disease is mainly characterized by keratinization of the skin and mucosa. When the silver-black fox suffers from this disease, it occurs nerve fiber myelin degeneration, female fox follicular degeneration and male fox convoluted seminiferous tubule epithelial degeneration, which leads to the reproductive function of the fox.

The clinical manifestations of young foxes and adult foxes are basically the same. generally, when vitamin An is deficient, clinical symptoms appear after 2-3 months. The early symptoms are neurological disorders, convulsions, tilting the head back, and the sick fox loses balance and falls down. The stress response of the sick fox is enhanced, and when it is stimulated by a small amount, it is highly excited, turns around the cage and wobbles its steps. The intestinal function of the young fox was damaged in varying degrees, showing symptoms of diarrhea, mixed with a large amount of mucus and blood in the feces, and sometimes symptoms of pneumonia, slow growth and slow tooth change.

[pathological anatomical changes] dead bodies are generally emaciated and anemic. Young foxes often have bronchitis and bronchitis. Young foxes also often find changes in gastroenteritis, ulcers in the stomach and stones in the kidney and bladder.

[diagnosis] the content of vitamin An in the blood of diseased foxes and the liver of dead animals was determined, and vitamin A could also be added to the diet for therapeutic diagnosis.

[prevention and treatment] to prevent the occurrence of this disease, we should first ensure the supply of vitamin An in the diet and pay attention to the supply of vegetables, fish and liver in the feed. To treat this disease, vitamin A can be added to the feed, the treatment amount is 5: 10 times the required amount, silver black fox and arctic fox daily each 3000~5000IU.

 
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