MySheen

Prevention and treatment of rickets in cultured masked palm civet

Published: 2024-11-05 Author: mysheen
Last Updated: 2024/11/05, Rickets is a nutritional and metabolic disease of young civets. It is a disease characterized by incomplete calcification, softness and deformation of bone tissue, growth retardation and heterophilia in diseased beavers due to vitamin D deficiency, insufficient or improper proportion of calcium and phosphorus. The disease usually occurs within 3 months.

Rickets is a nutritional and metabolic disease of young civets. It is a disease characterized by incomplete calcification, softness and deformation of bone tissue, growth retardation and heterophilia in diseased beavers due to vitamin D deficiency, insufficient or improper proportion of calcium and phosphorus. The disease mostly occurs in young beavers who are weaned within 3 months or early, especially when they are artificially fed, when they are lack of inorganic salt, vitamin D and protein in the diet and when they suffer from gastrointestinal disease. Adult beavers develop a similar disease called osteomalacia. The clinical features of congenital rickets are weakness, inability to stand, bending of forelimb bones, arched back, and even inability to suck milk. Acquired rickets shows a gradual development: first of all, the performance of excitement, ecstasy, like to gnaw on walls, wood and other filthy foreign bodies. Then the beaver has mental dementia, weakened digestive function, loss of appetite or abstinence, gradual weight loss, stagnation of growth, and rough coat. Sick beaver likes to lie down, or arch waist concave back, gait is strong, sometimes appear systemic ankylosing spasm, joint swelling, skull bulge, osteoporosis, dyspnea and so on. The resistance of the diseased beaver is reduced, and it is easy to have complications or systemic weakness, anemia, or even death. It is not difficult to diagnose according to age, management level, clinical symptoms, chronic process, growth retardation, paranoia, dyskinesia and bone changes. The levels of serum calcium and phosphorus also have diagnostic significance.

The main purpose of prevention and treatment is to supplement vitamin D, calcium and phosphorus. The following methods can be used to prevent and cure rickets: ① intravenous injection of 10% calcium gluconate 0.5ml / kg body weight, twice a day for 7 days. ② was injected intramuscularly with vitamin colloidal calcium solution 0.05ml / kg body weight once a day for 7 days. ⑧ oral calcium carbonate 5-1 g / time, once a day. ④ took orally vitamin D, 400ml 600 units / time, once a day for 7-10 days. ⑤ was injected subcutaneously or intramuscularly with cod liver oil (containing vitamin D800Mel 1000 units). The prevention of rickets is mainly to feed vitamin D and phosphorus, calcium-rich feed, each young beaver diet should be 100 units of vitamin D100. Commonly used bone meal to supplement calcium I and phosphorus, the content of calcium in the diet should not be less than 5%, and the ratio of calcium to phosphorus should be 1: 1, 2: 1.

 
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